Report Relating To
Physiologic Abnormalities
in Abductees
By Phillip S. Duke Ph.D
Copyright (c) 1999 Phillip S. Duke Ph.D. All Rights Reserved.
Introduction: Persons allegedly abducted by ET Aliens have been reported to experience the vitamin A deficiency induced abnormality Nyctalopia (night blindness) despite diets providing adequate Vitamin A (1). This non-dietary vitamin A deficiency may be due to the mechanism of implant mediated vitamin A destruction. Vitamin A is involved in steroidogenesis (biosynthesis of steroid hormones by the adrenal cortex) and evidence is presented for steroid hormonal deficiencies in abductees, presumably related to vitamin A deficiency.
Salt craving and anesthetic difficulties in abductees have also been previously reported (1). Salt craving is logically due to excessive urinary salt loss, due to deficiency of the adrenal steroid mineralocorticoid hormone Aldosterone, which functions primarily to reduce salt loss.
This Report briefly presents four abductee cases, one each in support of non-dietary Nyctalopia, low Aldosterone level, transient low adrenal glucocorticoid hormone level, and one case of evident deficiency involving the female sex hormone Estrogen. The Estrogen deficiency signs and symptoms were reversed by adding carrots and Vitamin E to the diet.
Adding carrots and vitamin E (which, both being antioxidants, has a sparing action on vitamin A) to the daily diet will safely and markedly increase body vitamin A, and by doing so in theory might thereby work towards reestablishment of normal hormonal levels, by facilitating steroidogenesis, and/or competing for implant mediated hormonal destruction.
Signs and symptoms of estrogenic deficiency in a pre-menopausal abductee taking Estradiol and vitamins without benefit, were promptly eliminated by adding carrots and vitamin E to her diet. This non-toxic inexpensive dietary addition may be of benefit to other alleged abductees with physiologic abnormalities. Persons adding the carrot vitamin E (CE) regimen to their daily diets are requested to inform me concerning procedures and results.
Background. In Derrel Sims' study of 250 alleged abduction cases about 50% of the subjects suffer from night blindness (Nyctalopia). About 90% of the female portion of the study suffer from Nyctalopia. All subjects exhibited a compulsion to consume salty substances and applied large amounts of common table salt to meals. All subjects showed abnormal reactions to procaine (dental) local anesthetics, in that either the normal amount was not sufficient or there were frank outright allergic reactions to these compounds (1). By my personal interviews with alleged abductees these reported results of Mr. Sims' studies have been essentially completely confirmed. These phenomena are as published on.
Visual Abnormalities. Nyctalopia (night blindness) is manifested by vision deficiency in dark adaptation. The person with night blindness typically cannot see adequately in dim light, where persons with normal vision can. Or when adaptation is still possible an abnormally long time is required. By making personal inquiries I have confirmed that females with a history of multiple abductions frequently cannot drive at night, crave salty foods, and may exhibit the described procaine anesthetic phenomena. One such alleged abductee informed me that night blindness was so common among her circle of alleged abductee friends it was assumed by them to be a female (sex linked genetic) trait. It is not. In theory genetically determined inability to employ vitamin A would be lethal. There are no genetic vitamin A deficiencies.
Nyctalopia is due to a deficiency in vitamin A. New vitamin A is constantly required (in small amounts) to produce the visual substance retinene as part of the normal visual cycle. Vitamin A is stored, primarily in the liver. Without adequate vitamin A intake the body stores eventually become depleted, and a deficiency occurs, resulting in Nyctalopia (2). Vitamin A is specific for the cure and prevention of the vitamin A deficiency states Nyctalopia and Hemeralopia (day blindness), and the severe vitamin A deficiency disease Xeropthalmia (3). Xeropthalmia refers to the more advanced clinical manifestations of severe Vitamin A deficiency which can lead to blindness, cell metaplasia, prominent in the lungs, and possibly even death.
Nyctalopia with adequate vitamin A intake. All the alleged abductees interviewed had more than adequate intake of vitamin A and precursors. One person I call Jane who by my evaluation is an abductee with implant stated she takes a Vitamin A tablet (5000 Units) and cod liver oil (another good source) daily. She also likes to eat carrots, broccoli and other good sources of carotenes, which are normally converted to Vitamin A. Her fat and protein (mostly chicken) intake (necessary for Vitamin A absorption and use respectively) were normal. Her digestion is normal and she is relatively healthy, though with Nyctalopia, salt craving, dental anesthetic problems, low resistance to infection, etc. There is no history of liver disease (carotene is converted to vitamin A in the liver). Therefore it is evident her Nyctalopia, salt craving, and anesthetic problems are not due to inadequate intake of Vitamin A. It appears that despite more than adequate intake the vitamin A in her body is not adequate to allow normal night vision.
Possible mechanism of vitamin A deficiency. It is reported that under black (long wavelength) ultraviolet (UV) light some implants fluoresced green (5). Vitamin A fluoresces a "characteristic green" (6). Therefore a possible simple mechanism explaining both vitamin A deficiency and implant fluorescence would be implant adsorption of vitamin A. Presumably followed by inactivation of vitamin A.
According to this concept, the implant induces Nyctalopia by inactivating vitamin A. Then despite apparently adequate intake, abductees with implants may have varying degrees of vitamin A deficiency caused by their implants. Warning- excessive amounts of vitamin A are dangerous. Its carotene precursors (carrots) are believed safe to eat in quantity.
There are alleged abductees without Nyctalopia, who drive satisfactorily at night, but also report frequently experiencing abnormal sensitivity to sunlight. At such times it hurts their eyes and produces tearing. This transient sensitivity might conceivably be due to a transient corresponding vitamin A deficiency. In my opinion such extreme visual sensitivity to sunlight, which might conceivably result over time in Hemeralopia (day blindness), is a more reliable indicator of possible abduction than is non-dietary Nyctalopia. Of course both conditions may have medical causes other than abduction, which causes must be ruled out before taking them for example as possible indicators of abduction.
Hormones of the Adrenal Cortex. The possibility exists that alleged abductees' common complaints of fatigue, susceptibility to infection, autoimmune type disorders, allergic reactions etc. may be related to sub-clinical vitamin A deficiency, which in addition may possibly result in deficiencies of hormones of the adrenal cortex. According to the literature "Vitamin A is implicated in steroidogenesis (steroid production) in the adrenal glands." (9). Implants may conceivably destroy these hormones as well as vitamin A. Then vitamin A deficiency may conceivably manifest in a variety of clinical problems related to decreased levels of the adrenal hormones, which include the mineralocorticoid Aldosterone (electrolyte regulation via sodium retention, sodium/potassium balance), and the glucocorticoids such as Cortisol (metabolic, antistress and antiallergic affects, and others.) (10).
Electrolyte Abnormalities. Salt craving is most commonly due to excessive loss of salt. Salt loss occurs in heavy sweating, due to elevated temperature, or in urinary excretion. The persons interviewed worked and lived in air conditioned spaces and were without fevers. Excessive salt loss into the urine is commonly associated with disease of the kidney or adrenal cortex. No history or evidence of kidney or adrenal cortex disease was presented. The adrenal cortex mineralocorticoid hormone Aldosterone normally regulates body salt retention by stimulating sodium ion retention. In uncompensated clinical Aldosterone deficiency the body suffers from markedly increased salt (sodium and chloride ion) loss into the urine, and there is also increased body potassium ion retention (4). This results in abnormally high potassium to sodium ratios in extracellular body fluids. This condition has been found among abductees (7).
In a recent case reported to me by Dr. Roger Leir a male abductee's 24 hour urine showed an Aldosterone level of 3.0 mcg/dL. The normal ranges are: Normal sodium intake 100- 200 mEq daily 6- 25 mcg/dL, High sodium intake greater than 200 mEq daily 0-6 mcg/dL, Low sodium intake less than 25mEq daily 17-44 mcg/dL Aldosterone. Sodium intake was normal, therefore the test result indicates low Aldosterone concentration.
There is the case of a mutilated cow that despite being on normal supplement and in Fall pasture, exhibited at autopsy an abnormally high extracellular potassium to sodium ratio, characteristic of abnormally low Aldosterone production, and also a very low liver Vitamin A level (8), so low that this animal very probably had Nyctalopia. According to my hypothesis the missing left ear had been implanted some time previously (11). Presumably the physiologic abnormalities were related to the implant.
Glucocorticoid Abnormalities. The concept that an implant may result in Aldosterone deficiency suggests the possibility that other hormones of the adrenal cortex may also be deficient.
The most clinically important such hormones are the glucocorticoids, which have numerous functions. These include influences on protein, carbohydrate and lipid metabolism, resistance to stress and an antiallergenic action. The glucocorticoids prevent histamine release. They therefore relieve the symptoms of asthma and delayed hypersensitivity reactions such as hives and serum sickness (see reference 10 p. 284). They are also of symptomatic benefit in...diseases in which allergic autoimmune reactions probably play a role (10).
Possible adrenal insufficiency after abduction. An adult male I call Brad who by my evaluation is a multiple abductee with implant, volunteered the information that after what he now believes was an early abduction experience with possible implantation, his sense of smell and taste were markedly enhanced. This puzzled him and he asked if I knew anything about it. I did not. Later I found in the literature that this phenomenon of markedly increased sensitivity of smell and taste is stated to occur as a result of marked adrenal insufficiency, presumably related to markedly reduced glucocorticoids (see reference (10) p. 280, Abstracts of the 44th meeting of the Endocrine Society). At this time his general health and sense of taste and smell are normal and he drives without difficulty at night. His transient sensory enhancement experience suggests that an early abduction was followed by a transient glucocorticoid deficiency.
Female Sex Hormone Deficiency. The sex hormones are produced in quantity by the gonads, and (in very small quantity) by the adrenal cortex. The possibility presents that an implant might produce sex hormone deficiency. A 57 year old (premenopausal) female I shall call Sue is by my evaluation a multiple abductee with implant. She suffered for many months from severe night hot flashes and sweats, preventing sleep, and amenorrhea (no menstruation). Along with the usual alleged abductee problems of Nyctalopia, salt craving and anesthetic difficulties. She was otherwise in generally good health, with a good diet supplemented by Centrum silver multivitamins/minerals once and St. John's wort 300 mg twice daily. Her gynecologist prescribed and she was taking Estrace (Estradiol) female sex hormonal supplement 2 mg daily for several months, without benefit. Provera (medroxyprogesterone) 2.5 mg was prescribed daily later.
Deficiency Correction by Carotene and Vitamin E. I suggested an added daily regimen of carrots (to safely supply vitamin A) along with vitamin E which has a sparing affect on vitamin E, helping it work. My idea was that the vitamin A in quantity might compete with female sex hormones for space on the implant, thereby hopefully sparing and allowing the female sex hormones to work. She initiated an additional daily dietary addition of 3 medium sized raw carrots and 800 units Vitamin E natural blend daily. Within a few days she reported the night hot flashes and sweats had stopped, allowing normal sleep, and normal menstrual bleeding began. Sue reported that "the bleeding was proportional to the carrot intake." The bleeding became excessive and she discontinued the carrots and Vitamin E. Her gynecologist performed a D&C and a biopsy under anesthesia. There were no anesthetic difficulties despite a history of severe such problems. The bleeding was controlled and the night hot flashes and sweats returned, though to a lesser degree than before, despite continued use of the Estradiol and Provera. The bleeding soon stopped and Sue returned to the full carrot regimen. The night hot flashes and sweats soon stopped and are now absent. Some cramping was present for a brief period. It is now also gone. The gynecological exam and biopsy results are normal. Sue is happy with the results of the carrot and vitamin E dietary addition and continues to employ it.
The foregoing cases suggest the possibility that adding carrots along with vitamin E to the daily diet, may be beneficial to abductees.
Concerning the concept that abductees (with physiologic abnormalities) may possibly be benefited by markedly increased vitamin A intake, if a person wants to try this, the safe way is by markedly increasing dietary intake of carotene precursors of vitamin A. The body safely converts these into vitamin A. Good sources are carrots, squash, broccoli, and other yellow and green vegetables and fruits. A minimal amount of fat or oil and quality protein (American chicken is recommended due to its yellow fat carotene content) are required for Vitamin A absorption and use. Vitamin E has a protective effect on Vitamin A, and should be taken to help enhance vitamin A's effects. Danger- excessive vitamin A itself is dangerous. For this reason I suggest that vitamin A not be taken. Just eat several carrots daily. And take vitamin E daily. Carrots can be eaten raw or cooked. Cooked carrots are more digestible.
Summary and Conclusions. The concept of alleged abductees with ET alien implants is evidently just too much for the scientific and UFO Journals. The MUFON UFO J. and The JSE have denied publication of this as they have of my previous work (11). In terms of hard science the four cases are too few. But- it is the best I can do. With what I have. Without funding of any kind, without access to laboratory facilities or peer publication, what are the possibilities? I could continue working in this area for untold years, amassing more data, which due to its (same) subject would also surely be denied publication. Or I can bring this information to the public's attention now, and learn the results. The suggested dietary addition of carrots and vitamin E is safe and inexpensive. Let interested alleged abductees try it and then let us see what happens. Valuable data will be obtained, one way or the other, and people may be helped now.
What I have done is essentially made an end run around the scientific and UFO establishments. Due to their unwillingness to help, I have now brought this information directly to the attention of those who may be interested in, and possibly benefit, from it. . All I ask is that information relating to use of the carrot and vitamin E (CE) dietary addition be brought to my intention. I am available to correspond with you and also to evaluate your possible abductee status. There is no charge, but if you can make any size donation please do so. I have no funding for this or any other work and all donations will be put to good use.
I am not licensed to practice medicine. If you desire medical assistance consult a physician without delay. I am not a physician. Should you have any questions, comments or related information please contact me.
Thank you. God Bless and Good Luck to us all.
Phillip S. Duke Ph.D.
Graduate USC Medical School with Ph.D. in Experimental Pathology.
2503 S. 47th St.,
Omaha, Ne 68106 USA
Author of the recent book titled "The AIDS-ET Connection"
Arcturus and Flatland books.
Research Associates include Dr. Roger Leir, Dr. Robert Koser, Mr. Harry
Jordan, Mr. John Buder, Mrs. Evelyn
Lorgen, Mr. Morgan Clements, and others.
(1). Leir, Roger K. The Alien and The Scalpel. Granite Publishing 1998 p.163.
(2). Cantarow, A. and Schepartz, B. Biochemistry. Saunders 1962 p.145-146.
(3). Ibid Reference (2). p. 148.
(4). Ibid reference (2). p. 719.
(5). Reference (1). p. 123.
(6). Merck Index 8th Edition Merck and Co., Inc. p.1111.
(7). Leir, Roger K. Private communication.
(8). National Institute for Discovery Sciences Las Vegas Nevada Final
Report June 1999.
(9). Pfeiffer, Carl C. Mental and Elementary Nutrients Keats Publishing
Inc. 1975 p. 198.
(10). Ganong, W.F. Medical Physiology 1963 Lange p. 263-294.
(11). Duke, Phillip S. The AIDS-ET Connection Cosmos Press 1999 Chapter 4.
Copyright (c) 1999 Phillip S. Duke Ph.D. All Rights Reserved.