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Allicin C And The Coronavirus
What Allicin C Does To Coronavirus Spike & Envelope Proteins

By Scott Perez
Research Scientist
Exclusive to Rense
4-8-20

You may recall I was the first person to calculate the Cannabis Receptor proper sequence relative to 3D folding Geometries  and that I am a GENBANK expert as I did my Doctoral Dissertation in Aging and Cancer. I gave you that exclusive live on  your radio program.  It is also on my website  www.canndetect.com  .

I have the genetic sequence of the Novel Coronavirus from the Fish market in Wuhan from NIH GENBANK.  
It is here  https://www.ncbi.nlm.nih. gov/nuccore/NC_045512

As far as I know, I am the first person to compute the Coronavirus Spike and the Envelope probabilities  of the viral components properly 3D folding right here...

The Cysteines are the target of Allicin in viral proteins or Bactria, fungi, yeast etc. infections.   It turns out in the Coronavirus, the Cysteines are mysteriously conserved and that is published  in the Journal of virology:

Importance of Conserved Cysteine Residues in the Coronavirus Envelope Protein

https://jvi.asm.org/content/ 82/6/3000

Further, if you look at the publication, you will find that the sequences match to the Novel Coronavirus.

The N-CoV virus has the conserved cysteines in the virus envelope.

Allicin C will create S-Allyl cysteine (below)

The ability of Allicin to cause the 3D geometry of the envelope to distort is greater because the Cysteines are conserved across the different types of coronaviruses per the GENBANK Homology analysis.

The spike mutates but each mutated sequence has a 1 in 2.6x10^3401 probability of existing. Multiple spike geometries are allowed in the coronavirus, so you have to have a major disruption in the protein folding by distorting the final 3D object. This is possible because there are 40 Cysteines in the Spike protein that are targeted by Allicin C.

The envelope probability is 1 in 2.65 x10^98. Itís smaller but with the conserved cysteines in the 40-44 region that likely have sulfide bonds, they are a better target for Allicin.
Since the envelope does not mutate like the spike and is conserved and required for function, it is a probable target for Allicin to Stop viral manufacturing in the cytoplasm of a human host cell.

Scott Perez
CannDetect, Patent Pending 62/993,481