Our Advertisers Represent Some Of The Most Unique Products & Services On Earth!

Bacteria In Strokes & Heart Disease
Reflections On The 'Cure' Of A
Paralyzed Stroke Victim

Copyright 2009 by Alan Cantwell, M.D.
Hi Jeff...
This communication is in response to a question you posed regarding the miraculous 'cure' of Russell McPhee, a 49 year old Australian man who had a devastating stroke and was paralyzed for more than two decades. After multiple injections of Botox into his spastic muscles, he was able to walk again. The details of this unusual recovery were published on Times Online ("Paralysed stroke victim 'cured' with Botox"
http://www.timesonline.co.uk/tol/news/uk/health/article6428858.ece), and posted on rense.com
Knowing my lifelong interest in bacteria as possible causative agents in certain chronic diseases and in cancer, Jeff Rense asked me: "This is a strange one. What might the link be between bacteria and a long term stroke?"
Before its well known use on wrinkles, Botox was used for stroke victims to counteract the muscles spasms. However, it is most unusual to get such a positive response after so many years, as
in McPhee's case.
Interestingly, Botox is Botulinum toxin, a neurotoxic protein produced by the bacterium Clostridium botulinum. It is the most toxic protein known. The intense forced relaxation of his
fibrotic and contracted muscles (along with physical therapy) was the key to initiating his 'cure.'
Professor John Olver, one of Australia's top stroke experts, said: "We use Botulinum toxin routinely for patients with spasticity
which has been caused by stroke, brain damage or heart disease.
But we use it very early on, usually within weeks of a stroke, to prevent the spasticity from becoming a problem. Sometimes the spasticity is so severe we inject those muscles with botulinum
toxin, which relaxes the muscles enough to allow a physiotherapist
to strengthen and stretch them. It is unfortunate that this patient had to wait for 20 years and extremely unusual that his treatment was so successful after being immobile for so long. But he's very fortunate that his muscles are strong enough to allow him to be able to walk."
Could bacteria play a role in initiating the stroke, as Jeff Rense asks? Can bacteria also play a role in chronic muscle disease, wasting, contractures, and fibrosis?
I will attempt to present some thoughts (as well as some published data) that might give a clue to the possible involvement of
bacteria in strokes and in the recovery of this long-paralyzed man by use of Botox injections.
First, there is more and more evidence that bacteria (and viruses) may play a heretofore unrecognized role in chronic illness.
Second, there is evidence that human blood is not sterile. On the contrary, all blood is infected with bacteria. A stroke in the form of a hemorrhage pours blood into the affected areas of the brain and damages the brain cells and nerve connections. A stroke in the form of a clot does not allow blood to get to the cells.
And thirdly, in many people, and particularly in the elderly, there is an intimate connection between stroke, heart disease, and chronic hypertension (high blood pressure). In addition,
prolonged hypertension frequently leads to kidney disease. If there is a bacterial connection to a stroke, there should also be some evidence to link bacteria to heart disease, to hypertension and to kidney disease. Whether this particular Australian has hypertension, kidney disease or heart disease is not mentioned in the report.
Bacteria and chronic muscle disease
By Googling "bacteria + muscle disease", one can easily review the evidence for bacteria (in the form of pleomorphic cell wall deficient bacteria, mycoplasma) in certain chronic diseases in which arthritic symptoms, muscular weakness and fibrotic changes take place over time. Some examples are rheumatoid arthritis,
Lyme disease, chronic fatigue syndrome, etc. Some patients with these diseases appear to be helped by longterm antibiotic therapy and other modalities, as proposed by the controversial "Marshall Protocol."
My own autopsy research into the "acid-fast bacteria" that
cause systemic scleroderma convinced me years ago that these bacteria were implicated in the muscle, cardiac, and kidney
abnormalities that accompany the hardening and fibrosis of the skin in fatal cases. (Figure 1 shows round tiny granular coccoid-appearing bacteria in the deep portion of the skin in scleroderma.) For more details, Google: "alan cantwell" + scleroderma.
Bacteria and the blood
Recent research, based on molecular biology, indicates that 90% of the cells of the human body are bacteria cells!! Despite this amazing new discovery, most physicians do not believe bacteria are involved in any way with the major diseases (heart disease, stroke and cancer) that kill most of us. In addition, most physicians
believe that healthy blood is sterile, that is, free of bacteria.
In 2007, rense.com posted an article of mine entitled: "All human blood is infected with bacteria" (http://www.rense.com/general77/dblooder.htm). In a series of papers from 1972-1979, the late Guido Tedeschi and his colleagues at the University of Camerino in Italy presented remarkable findings indicating universal infection of the blood with staphylococcus-like and streptococcal-like microbes.
In 1977 Domingue and Schlegel confirmed "the existence of a novel bacteriologic system" in the blood. They cultured staphylococcal-like bacteria and filamentous cocco-bacillary forms from 71% of the blood specimens from ill patients; and from 7% of supposedly healthy people. These pleomorphic (variation in size and shape) bacteria grew out of round complex "dense bodies" and developed into "ordinary bacteria." The authors concluded: "These organisms may represent an adaptation of certain bacteria to life in the blood." Their full report, which contains pictures (full-screen) of the bacteria grown from human blood, is online at: http://www.pubmedcentral.nih.gov/pagerender.fcgi?artid=421412&pageindex=1#page
In the 1990's microbiologists Phyllis E Pease and Janice Tallak termed these blood bacteria as "the human bacterial endoparasite." Finnish researchers Kajander et al. describe them as "novel bacteria-like particles," which are staphylococcal-like. Like
viruses, these tiny bacterial forms were able to pass through bacterial filters, and were exceedingly difficult to culture. The Finnish team called them "nanobacteria" and proposed a tentative name for the novel agent: Nanobacterium sanquineum.
In 2002 McLaughlin et al. presented a study entitled "Are there naturally occurring pleomorphic bacteria in the blood of healthy humans?" The researchers were surprised to discover bacteria in the blood "since it is generally acknowledged that the blood stream in healthy humans is a sterile environment, except when there is a breach in the integrity of the tissue membranes."
In addition, this blood research confirms the findings of Gunther Enderlein (1872-1968), Wilhelm Reich (1897-1957) ), Raymond Royal Rife (1888-1971), Virginia Livingston (1906-1990), Gaston Naessens (1924- ) and others whose research can be easily googled.
Bacteria and hypertension
Because cryptic bacterial infection of the blood is not accepted by most scientists, there have been no studies correlation infection with hypertension. However, a just-published report (May 16, 2009) suggests that high blood pressure could be caused by a common virus, known as the cytomegalovirus (CMV), a common viral infection affecting between 60 and 99 percent of adults worldwide. (http://www.sciencedaily.com/releases/2009/05/090514221915.htm) Previous studies determined that the CMV virus was linked to the development of atherosclerosis, the hardening of the heart's arteries. This new study brought together a team of researchers from a variety of disciplines ­ infectious diseases, cardiology,
allergy and pathology ­ to look more closely at the issue.
Bacteria and the Heart
In an autopsy study published in 1965, my mentors Virginia (Wuerthele-Caspe) Livingston and Eleanor Alexander-Jackson first showed acid-fast bacterial infection of the heart muscle, in the coronary blood vessels and in the aorta, in a paper entitled "Mycobacterial forms in myocardial vascular disease." Later, they proposed a vaccine to combat the bacteria they observed not only in heart disease, but in cancer and degenerative disease as well. A summary of their research can be read by googling: U.S. Patent# 4692412). Figure 2 shows bacteria in the heart muscle at autopsy in a fatal case of AIDS. Figure 3 shows bacteria in the heart muscle from an autopsied case of lymphoma (mycosis fungoides).
Most recently, in a BBC news report on May 25, 2009, there has been a renewed interest in bacteria and heart disease. A scientific team, headed by Arne Schaefer at the University of Kiel, found a common gene mutation in patients with periodontitis and heart attack patients. There were also similarities between the bacteria found in the oral cavity and those in coronary plaques. One theory is that the bacteria involved in gum disease trigger a low grade inflammatory response throughout the body, prompting changes in the arteries leading to strokes and heart attacks. Another possibility is that the bacteria disturbs the way blood vessels dilate directly, since some bacteria can enter the bloodstream. (http://news.bbc.co.uk/2/hi/health/8063512.stm).
Bacteria and strokes
Only in the past few years has there been mild interest in investigating the role of bacteria in the pathogenesis of strokes. A Google search using key words "bacteria" + "strokes" leads to numerous web articles, such as "Ulcer bacteria linked to stroke', "Bacteria in mouth could predict strokes", "Antibiotics help prevent strokes", etc.
Obviously much more research needs to be done on the precise role of bacteria in stroke formation. In my view, if bacteria are
universally present in the blood, then they might play an important role in stroke formation.
Bacteria in kidney disease
As mentioned, chronic hypertension can lead to kidney disease. The role of bacteria (particularly pleomorphic cell wall deficient bacteria) in renal and urinary disease has been extensively studied and pioneered by microbiologist Gerald Domingue, author of Cell Wall Deficient Bacteria (1982). One can go to the PubMed website to view 38 published of his papers, many concerning
cryptic bacterial infection of the blood, the kidney and the urinary tract. There is also an extensive bio on Domingue found on Wikipedia. Tragically, there is little interest in studying the role of bacteria in kidney disease. Figure 4 shows bacteria detected in the kidney in a fatal case of systemic scleroderma. Note the similarity of these bacterial forms to the bacterial forms depicted in the heart.
Perhaps the biggest reason why bacteria are ignored in human disease, such as heart disease, cancer, and chronic disease, is that these microbes are so ubiquitous. Everyone carries them, both in sickness and in health. And doctors have a hard time comprehending that the same bacteria present in a healthy individual can be similar, if not identical, to the bacteria that are found in people who are not healthy.
However, almost all healthy people die of diseases, such as cancer and heart disease or kidney disease, or suffer a stroke. In that sense, if one lives long enough, all healthy people eventually become diseased people. Livingston was fond of saying "the microbe" was both the giver and the taker of life.
I am grateful Jeff Rense asked his question about the stroke and miraculous 'cure' of Russell McPhee. It gave me the courage to write about bacteria in diseases that most physicians do not consider as bacterial-induced illnesses. However, I think it is fair to say that we still know very little about the bacteria we carry in our bodies, and the trouble they can cause as we age.
One final point: Why did a bacterial-produced Botox product start a healing? In my view, I suspect the paralytic product forced a "breaking up" of the tight, constricted muscle. This allows better blood flow to the muscle. This, along with the message, has a healing and energy-renewal quality which "flushed out" the bacteria in the muscle. I assume this re-creates some new "channels" between the brain and the muscles. I am not a neurologist or a physiologist, but this is how I see it. Figure 5 show the tiny round bacterial forms in the skeletal muscle of a fatal case of AIDS, a disease that is well-known to cause weakness and "wasting" of the muscles.
Despite the heretical aspects of this paper, I trust some researcher somewhere will take an interest in these newer findings and attempt to prove or disprove them.
Domingue GJ, Schlegel JU.Novel bacterial structures in human blood: cultural isolation. Infect Immun. 1977 Feb;15(2):621-7.
Kajander EO, Tahvanainen E, Kuronen I and Ciftcioglu N.
Comparison of Staphylococci and Novel Bacteria-Like Particles from Blood. Zbl. Bakt. Suppl. 26, 1994.
McLaughlin RW, Vali H, Lau PC, Palfree RG, De Ciccio A, Sirois M, Ahmad D, Villemur R, Desrosiers M, Chan EC. Are there naturally occurring pleomorphic bacteria in the blood of healthy humans? J Clin Microbiol. 2002 Dec;40(12):4771-5.
Nikkari S, McLaughlin IJ, Bi W, Dodge DE , Relman DA. Does blood of healthy subjects contain bacterial ribosomal DNA? J Clin Microbiol. 2001 May;39(5):1956-9.
Pease PE, Tallack JE. A permanent endoparasite of man. 1. The silent zoogleal/symplasm/L-form phase. Microbios. 1990;64(260-261):173-80.
Tedeschi GG, Di Iorio EE. Penetration and interaction with haemoglobin of corynebacteria-like microorganisms into erythrocytes in vitro. Experientia. 1979 Mar 15;35(3):330-2.
Tedeschi GG, Bondi A, Paparelli M, Sprovieri G. Electron microscopical evidence of the evolution of corynebacteria-like microorganisms within human erythrocytes. Experientia. 1978 Apr 15;34(4):458-60.
Tedeschi GG, Amici D, Sprovieri G, Vecchi A. Staphylococcus epidermidis in the circulating blood of normal and thrombocytopenic human subjects: immunological data. Experientia. 1976 Dec 15;32(12):1600-2.
Tedeschi GG, Amici D. Mycoplasma-like microorganisms probably related to L forms of bacteria in the blood of healthy persons. Cultural, morphological and histochemical data.Ann Sclavo. 1972 Jul-Aug;14(4):430-42.
Wuerthele-Caspe (Livingston) V, Alexander-Jackson E. Mycobacterial forms in myocardial vascular disease. J Amer Med Wom Assoc. 1965 (20):449-452.
Alan Cantwell M.D. is retired dermatologist. He is the author of The Cancer Microbe: The Hidden Killer in Cancer, AIDS, and Other Immune Diseases, and Four Women Against Cancer: Bacteria, Cancer and the Origin of Life, both published by Aries Rising Press, PO Box 29532, Los Angeles, CA 90029 (www.ariesrisingpress.com). His books are available from Amazon.com and via Book Clearing House at 1-800-431-1579.
Email address: alancantwell@sbcglobal.net
Alan Cantwell M.D.
author of, AIDS & The Doctors of Death and Queer Blood
Donate to Rense.com
Support Free And Honest
Journalism At Rense.com
Subscribe To RenseRadio!
Enormous Online Archives,
MP3s, Streaming Audio Files, 
Highest Quality Live Programs


This Site Served by TheHostPros