- From John DeShiro
- Regarding the post on Chemtrail Lab Analysis - Virulent
- US Code Title 15 Section 1520 was repealed in 1997...
- and replaced with1520a
- ...which contains much of the same wording with a different
- If you first take a tour to www.wildlandsproject.org/index.shtml,
click on the site map, then go to page 3 you will find where it states
that they have to reduce the size of the human population by 'humane' means.
This got me to wondering if they thought that chemical spraying was in
their opinion a 'humane' means to reduce the population. I don't know if
you have heard of William Thomas, but he has a lot of info on the chemtrails.
His website is www.islandnet.com/~wilco/. If you click on issue # 6 you
can get a lot of information. He has posted on his site BIOWARFARE TEST
ON THE AMERICAN PEOPLE.
- This states that the recently revised U.S. Code Title
50, Section 1520 states that the Secretary of Defense may conduct tests
or experiments "involving the use of a chemical agent or biological
agent on a civilian population" if they are related to research activity.
The law also stipulates that biowarfare test can be carried out on Americans
only if Congress is notified 30 days in advance, and "only if informed
consent to the testing was obtained from each human subject in advance
of the testing on that subject."
- There is plenty of pattern and precedent to suggest that
clandestine biowarfare experiments are routinely pacticed on the American
public without their informed consent. Two Congressional investigations
in1977 and 1994 - and recently declassified British defense documents -
detail 50 years of "open air" testing that used ships and spray-equipped
aircraft to spread biological warfare simulaants on hundreds of cities
across the U.S., Canada and the U.K. There is more to this article. I just
wanted to give you some of the info because could spend the rest of the
day typing this stuff up.
- So far some of the chemicals that were collected from
the chemtrail spray and lab tested is:
- 1. Bacilli & Molds 2. Pseudomonas Aeruginosa 3. Pseudomonas
Florescens 4. Bacilli Amyloliquefaciens 5. Streptomyces 6. Enterobacteriaceae
7. Serratia Marcscens 8. Human White Blood Cells 9. A restricter enzyme
used in research labs to snip and combine DNA 10. Enterobacter Cloacae
11. Other bacilli and other toxic molds capable of producing heart disease
and meningitis, as well as acute upper respiratory and gastrointestinal
distress. 12. JP-8 Jet Fuel = Ethylene Dibromide
- # 2. Pseudomonas Aeruginosa = Respiratory tract infection
by the ubiquitous bacterium. Cancer and burn patients also commonly suffer
attack from this organism. Unlike other bacteria that reside in the enviroment,
P. aeruginosa has a remarkable capacity to cause disease. Pseudomonas has
the ability to adapt and thrive in many ecological niches, including humans.
Once infections are established, P. aeruginosa produces a number of toxic
proteins which cause not only extensive tissue damage, but also interfere
with the human immunesystem's defense mechanisms. These proteins range
from potent toxins that enter and kill host cells at or near the size of
colonization todegradative enzymes that permantly disrupt the cell membranes
and connective tissues in various organs. P. aeruginosa successfully colonizes
the respiratory tract. One reason is that it produces a highly protective
capsule made of the mucoid polysaccharide alginate. This allows the bacteria
to resist engulfment by immune systemcells and better adhere to the lining
of the lungs. It is likely that antibiotics cannot effectively eradicate
Pseudomonas from the lungs because of this protective capsule. In addition,
some Pseudomonas strains can inactivate the drugs that threaten them by
using enzymes to modify the drug.
- # 7. Serratia Marcescens is a significant opportunistic
human bacterial pathogen. This microorganism has been shown to be the cause
of many life-threatening diseases such as pneumonia, meningitis and endocarditis.
It is one of the major causes of hospital-acquired infections. The seriousness
of a S. marcescens infection is compounded by the fact that it is very
resistant to most commonly used antibotics, thus making treatment difficult.
In this study one of the factors contributing to the antibiotic resistance
of S. marcescens will be examined. In order for an antibiotic to kill or
inhibit growth of bacteria it must penetrate the outer surface or membrane
and enter the bacterial cell which is very difficult. I hope some of this
helps you to better understand what we are fighting against.
- To learn more, contact: www.spiralintoit.com
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