- Hello, Jeff - I am 100% sure that Mrs. Adablo's work,
at Tyson, where she worked on the assembly line slaughtering cattle, most
assuredly led to her death from a form of CJD/Mad Cow Disease.
-
- There really needs to be an epidemiological follow up
of workers who work in slaughtering plants. My guess is Mrs. Adablo is
not the first worker. I would bet the farm that if such a health followup
were carried out that we would also see many deaths from 'Alzheimers' Disease
as well as Mad Cow.
-
-
- Patty
-
- Patricia A. Doyle DVM, PhD Bus Admin, Tropical Agricultural
Economics Univ of West Indies Please visit my "Emerging Diseases"
message board at:http://www.emergingdisease.org/phpbb/index.php Also
my new website: http://drpdoyle.tripod.com/ Zhan le Devlesa tai
sastimasa Go with God and in Good Health
-
- From hshields@worldpath.net
-
- Need Spanish translation . . .
- http://www.ranchers.net/forum/post-454444.html
-
- comment . . .
-
- I see Linda Andablo died on February 6, 2010 - quite
a while ago - I wonder what has gone on behind the scenes between
2/6 and now . . . . . they (USDA, etc.) have had a long time to make
mischief !
-
- do you know if any of the TSE experts (including those
in UK) are or have participating in an autopsy or examining
sections of the brain? Do you know if her brain is being examined for
NV CJD ? Her employment at the slaughterhouse particupating in euthanasia
and exposed to "brain mist" and potentially prion infected brains,
spinal cords, etc. creates quite a problem for the USDA spin factory .
. . ? They cannot admit she had nv CJD because it would acknowledge
infected animals are being processed at abattoirs . . . . but she is too
young for sporadic . . . . .
-
- Helane
-
-
- http://www.recordandoalinda.com/index.php?option=com_content&view=article
- &id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8
-
-
- Irma Linda Andablo CJD Victim, she died at 38 years old
on February 6, 2010 in Mesquite Texas.She left 6 Kids and a Husband.The
Purpose of this web is to give information in Spanish to the Hispanic community,
and to all the community who want's information about this terrible disease.-
-
- Physician Discharge Summary, Parkland Hospital, Dallas
Texas
-
- Admit Date: 12/29/2009 Discharge Date: 1/20/2010
- Attending Provider: Greenberg, Benjamin Morris; General
Neurology Team: General Neurology Team
-
- Linda was an Hispanic female with no past medical history
presents with 14 months of incresing/progressive altered mental status,
generalized weakness, inability to walk, loss of appetite, inability to
speak, tremor and bowel/blader incontinence.She was, in her usual state
of health up until February, 2009, when her husbans notes that she began
forgetting things like names and short term memories. He also noticed mild/vague
personality changes such as increased aggression. In March, she was involved
in a hit and run MVA,although she was not injured. The police tracked her
down and ticketed her. At that time, her son deployed to Iraq with the
Army and her husband assumed her mentation changes were due to stress over
these two events. Also in March, she began to have weakness in her legs,
making it difficult to walk. Over the next few months, her mentation and
personality changes worsened, getting to a point where she could no longer
recognized her children. She was eating less and less. She was losing more
weight. In the last 2-3 months, she reached the point where she could not
walk without an assist, then 1 month ago, she stopped talking, only making
grunting/aggressive sounds when anyone came near her. She also became both
bowel and bladder incontinent, having to wear diapers. Her '"tremor'"
and body jerks worsened and her hands assumed a sort of permanent grip
position, leading her family to put tennis balls in her hands to protect
her fingers.
-
- The husband says that they have lived in Nebraska for
the past 21 years. They had seen a doctor there during the summer time
who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood
disorder. However, the medications did not help and she continued to deteriorate
clinically. Up until about 6 years ago, the pt worked at Tyson foods where
she worked on the assembly line, slaughtering cattle and preparing them
for packaging. She was exposed to brain and spinal cord matter when she
would euthanize the cattle. The husband says that he does not know any
fellow workers with a similar illness. He also says that she did not have
any preceeding illness or travel.
-
- What is Creutzfeldt-Jakob Disease?
-
- Creutzfeldt-Jakob disease (CJD) is a rare, degenerative,
invariably fatal brain disorder. It affects about one person in every one
million people per year worldwide; in the United States there are about
200 cases per year. CJD usually appears in later life and runs a rapid
course. Typically, onset of symptoms occurs about age 60, and about 90
percent of patients die within 1 year. In the early stages of disease,
patients may have failing memory, behavioral changes, lack of coordination
and visual disturbances. As the illness progresses, mental deterioration
becomes pronounced and involuntary movements, blindness, weakness of extremities,
and coma may occur.
- There are three major categories of CJD:
-
- In sporadic CJD, the disease appears even though the
person has no known risk factors for the disease. This is by far the most
common type of CJD and accounts for at least 85 percent
-
- In hereditary CJD, the person has a family history of
the disease and/or tests positive for a genetic mutation associated with
CJD. About 5 to 10 percent of cases of CJD in the United States are hereditary.
- In acquired CJD, the disease is transmitted by exposure
to brain or nervous system tissue, usually through certain medical procedures.
There is no evidence that CJD is contagious through casual contact with
a CJD patient. Since CJD was first described in 1920, fewer than 1 percent
of cases have been acquired CJD.
-
- CJD belongs to a family of human and animal diseases
known as the transmissible spongiform encephalopathies (TSEs). Spongiform
refers to the characteristic appearance of infected brains, which become
filled with holes until they resemble sponges under a microscope. CJD is
the most common of the known human TSEs. Other human TSEs include kuru,
fatal familial insomnia (FFI), and Gerstmann-Straussler-Scheinker disease
(GSS). Kuru was identified in people of an isolated tribe in Papua New
Guinea and has now almost disappeared. FFI and GSS are extremely rare hereditary
diseases, found in just a few families around the world. Other TSEs are
found in specific kinds of animals. These include bovine spongiform encephalopathy
(BSE), which is found in cows and is often referred to as "mad cow"
disease; scrapie, which affects sheep and goats; mink encephalopathy; and
feline encephalopathy. Similar diseases have occurred in elk, deer, and
exotic zoo animals.
-
- What are the Symptoms of the Disease?
-
- CJD is characterized by rapidly progressive dementia.
Initially, patients experience problems with muscular coordination; personality
changes, including impaired memory, judgment, and thinking; and impaired
vision. People with the disease also may experience insomnia, depression,
or unusual sensations. CJD does not cause a fever or other flu-like symptoms.
As the illness progresses, the patients' mental impairment becomes severe.
They often develop involuntary muscle jerks called myoclonus, and they
may go blind. They eventually lose the ability to move and speak and enter
a coma. Pneumonia and other infections often occur in these patients and
can lead to death.
-
- There are several known variants of CJD. These variants
differ somewhat in the symptoms and course of the disease. For example,
a variant form of the disease-called new variant or variant (nv-CJD, v-CJD),
described in Great Britain and France-begins primarily with psychiatric
symptoms, affects younger patients than other types of CJD, and has a longer
than usual duration from onset of symptoms to death. Another variant, called
the panencephalopathic form, occurs primarily in Japan and has a relatively
long course, with symptoms often progressing for several years. Scientists
are trying to learn what causes these variations in the symptoms and course
of the disease.
-
- Some symptoms of CJD can be similar to symptoms of other
progressive neurological disorders, such as Alzheimer's or Huntington's
disease. However, CJD causes unique changes in brain tissue which can be
seen at autopsy. It also tends to cause more rapid deterioration of a person's
abilities than Alzheimer's disease or most other types of dementia.
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