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Dental/Medical Mad
Cow/CJD Transmission Risks

Compiled By Jeff Rense

Invasive 'Sterilized' Re-Usable Medical/Dental Instruments As Modes
Of CJD Transmission
CJD/TSE Transmission Via Dental Instruments
Scientists Warn Of Mad Cow/CJD Spread In Dental And Surgical Procedures
Precautionary Advice Given To Dentists On vCJD
Master Dentist Falls Victim To vCJD, Dies
#8 Call For New National Dental/Medical Sterile Guidelines
US Mad Cow Strain More Virulent To Humans Than UK
Draft minutes of the SEAC 95 meeting (426 KB) held on 7
December 7, 2006 are now available.
January 18 2007
64. A member noted that at the recent Neuroprion meeting, a study was presented showing that in transgenic mice BSE passaged in sheep may be more virulent and infectious to a wider range of species than bovine derived BSE.
Other work presented suggested that BSE and bovine amyloidotic spongiform encephalopathy (BASE) MAY BE RELATED. A mutation had been identified in the prion protein gene in an AMERICAN BASE CASE THAT WAS SIMILAR IN NATURE TO A MUTATION FOUND IN CASES OF SPORADIC CJD.
'Sporadic' CJD - The Big Lie
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet- mg&T=0&P=25276
Steady Rise In Sproradic CJD In US - Unknown Strain
Transmission Of Italian Atypical BSE (BASE) In Mouse
Models Qingzhong Kong, Ph.D., Assistant Professor, Pathology, Case Western Reserve University
Bovine Amyloid Spongiform Encephalopathy (BASE) is an atypical BSE strain discovered recently in Italy, and similar or different atypical BSE cases were also reported in other countries. The infectivity and phenotypes of these atypical BSE strains in humans are unknown.
In collaboration with Pierluigi Gambetti, as well as Maria Caramelli and her co-workers, we have inoculated transgenic mice expressing human prion protein with brain homogenates from BASE or BSE infected cattle. Our data shows that about half of the BASE-inoculated mice became infected with an average incubation time of about 19 months; in contrast, none of the BSE-inoculated mice appear to be infected after more than 2 years.
*** These results indicate that BASE is transmissible to humans and suggest that BASE is more virulent than classical BSE in humans. ***
Additional Data:
1. Diagnosis and Reporting of Creutzfeldt-Jakob Disease
Singeltary, Sr et al. JAMA.2001; 285: 733-734.
http://jama.ama-assn.org/http://www.neurology.org/cgi/eletters/ 60/2/176#535
15 November 1999
US Scientists Should Be Concerned With A CJD Epidemic In The US
MARCH 26, 2003
RE-Monitoring The Occurrence Of Emerging Forms
of Creutzfeldt-Jakob Disease in the United States
Terry S. Singletary
I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic
CJDs in the USA are type 2 PrPSc?
Copyright © 2003 Published by Elsevier Ltd.
Tracking Spongiform Encephalopathies In North America
By Xavier Bosch
Available online 29 July 2003.
Volume 3, Issue 8, August 2003, Page 463
http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/ fulltext
http://download.thelancet.com/pdfs/journals/1473-3099/ PIIS1473309903007151.pdf
see history of cjd questionnaire
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